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Vitamin D from Sunlight for Immune Restoration - may be a co-factor that enhances Black Seed’s benefits

Black Seed powder improves blood cancer markers in a local patient

Conrad LeBeau

On April 27, a local female cancer patient (Patient D) told Keep Hope Alive that she has been treated for bone cancer with chemotherapy since July of 2014. Patient D who walks with a cane and is in her 50's said that she has osteo-carcinoma (bone cancer). She said her oncologist has been tracking her progress with a blood test called CA 27.29. Last summer this marker was 103 and with chemotherapy it has dropped to 62.

However, on April 7th, she had added 3 Black Seed powder capsules taken twice a day to her daily regimen. A follow up test 14 days later showed the CA 27.29 marker had dropped from 62 to 47. This is an encouraging sign indicating that the cancer activity was declining. In, they report -

"CA 27.29 is a mucus-containing protein that is produced by the MUC-1 gene."

Breast cancer cells and some other types of cancers will shed copies of the CA 27.29 protein into the bloodstream. When CA 27.29 numbers decline it indicates the cancer is less active or declining and that the treatment is probably working.

She, (patient D), continues using the Black Seed powder along with the chemotherapy and will have a follow up test in another 2 weeks. She told me she also started taking 800 mcg daily of Phytosel - a plant based selenium distributed by about the same time. Selenium's role in the prevention and treatment of cancer is well documented with numerous scientific citations in the Immune Restoration Handbook.

However, in our conversation, she said she thought it was the Black Seed capsules that contributed to the decline in her cancer blood markers leaving her hopeful that more good news will follow.

June 1st update on Patient D

She told me last week that her CA 27.29 test now indicates it has declined to 45 down from 47 last month. It was 103 last year, and gradually declined to 62. When she added Black Seed capsules early in April, it dropped to 47 within 3 weeks. She also undergoes periodic treatments with low dose chemotherapy along with several other protocols. She says she also takes 10,000 i.u. daily of vitamin D3.

A six-month Black Seed mono-therapy case report from Europe fails to duplicate the Nigerian case studies

A case report from Europe, (relayed by Denis of Croatia last month) where the HIV meds were stopped for 6 months to use Black seed as a mono-therapy failed to duplicate the results of the Nigerian studies by Dr. Onifade et al. After 6 months of using black seed powder mixed with honey (2 or more teaspoons three times a day) CD4 counts were lower than baseline while the viral load for HIV had increased. This occurred even though the person reported he was asymptomatic, had more energy, better lipid blood levels, than before using the black seed.

Here is an excerpt – May 26th email from Denis of Croatia -

My friend has been taking black seed honey (made by Onifade's recipe) and his new results are worse than ones from the beginning. Somewhere about Christmas: CD4 1100 and Viral Load about 800. This is the time he stopped the meds and started using nigella powder honey mixture. New results from May 26: CD4 300 and VL 150 000

According to his email, his friend has gone back to using the HIV meds. A second case from the United States using Black Seed as a mono therapy for 6 months also had similar disappointing results. The disparities in results obtained by Dr. Onifade and his cohorts in Nigeria and failures to duplicate his impressive results in a few isolated cases here in the United States and Europe raises some questions -

1. Was the strain of HIV in Nigeria HIV-2, known to be a weaker strain then HIV-1?

2. Denis of Croatia noticed an interesting anomaly about the Nigerian cases. The viral load for the HIV patients was surprisingly low for not being on any anti viral meds or remedies. Were their dietary, genetic or environmental factors responsible for these anomalies?

3. Was the crucial factor for the success of Black seed powder in the Nigerian studies that the Nigerian patients spent significantly more time outdoors with their skin exposed to the sun resulting in higher than average Vitamin D levels? [With a plethora of scientific research linking vitamin D to improved immune function, this seems like me a plausible path to pursue.]

The Sun, Vitamin D, and Immunity

Does Black Seed therapy also require the person to have significant daily exposure to the sun to receive the benefits of using Black Seed? Could the sun have a significant role and be a co-factor in strengthening and balancing the immune response to HIV?

In Nigeria, a country closer to the Equator than is either the United States or Europe, Dr. Onifade’s test patients probably received significantly more exposure to the sun than had they lived in either the United States or Europe.

Was daily exposure to the sun a factor that helped produce the results reported in the scientific articles he co-authored? Would increasing the levels of vitamin D from exposure to more sunlight or supplementation with vitamin D3 help the immune system in its battle against HIV as it has against other diseases like hepatitis B?

Finally, did increased daily exposure to sunlight improve the immune response of Onifade's patients even from the onset of his studies where the base line of HIV was surprisingly low - usually less than 50,000 (as measured by pcr)?

The following abstract published earlier this year raises the possibility that daily sunlight exposure was a co-factor in the Nigerian patients that was missing in the random Black Seed test cases here in the United States and Europe. The following abstract is reprinted in its entirely. [We have not yet obtained the full text article of this study.]

An update on the association of vitamin D deficiency with common infectious diseases

Authors - Watkins RR1, Lemonovich TL, Salata RA. Can J Physiol Pharmacol. 2015 May;93(5):363-8. doi: 10.1139/cjpp-2014-0352. Epub 2015 Jan 26.


"Vitamin D plays an important role in modulating the immune response to infections. Deficiency of vitamin D is a common condition, affecting both the general population and patients in health care facilities. Over the last decade, an increasing body of evidence has shown an association between vitamin D deficiency and an increased risk for acquiring several infectious diseases, as well as poorer outcomes in vitamin D deficient patients with infections.

"This review details recent developments in understanding the role of vitamin D in immunity, the antibacterial actions of vitamin D, the association between vitamin D deficiency and common infections (like sepsis, pneumonia, influenza, methicillin-resistant Staphylococcus aureus, human immunodeficiency virus type-1 (HIV), and hepatitis C virus (HCV)), potential therapeutic implications for vitamin D replacement, and future research directions."


All of the test cases in the Onifade Black Seed studies had unusually low level of HIV infection at the onset of using this remedy. Published studies on the prevalence of HIV-1 and HIV-2 in Nigeria indicate they are about equal with slightly more HIV-1 in the general population. It is plausible to that some of the Nigerian patients had HIV-1 and others HIV-2 and a few may have had both.

Considering how much closer to the Equator Nigeria is than Europe or the United States, the Nigerian patients daily exposure to sunlight was probably greater than the individual test cases here in the United States and Europe. Because the research on vitamin D is so exhaustive, and this research supports the need for more vitamin D in many subsets of illnesses, I refer you to this link at Pubmed at the National Library of Medicine.

Pubmed Articles on Vitamin D and immunity

Here you can read on the association between vitamin D and immunity. It has links to 2301 scientific articles.

Update June 8th. I located the following article this morning on the status of vitamin D levels in Nigerian children. Although this article shows that Nigerian children spend an average of 8.3 hours a day outdoors, all have vitamin D levels in excess of 10 ng/ml.

Conclusion - in spite of their dark skin, African children in this study were not deficient in Vitamin D. This also provides proof that in spite of their pigmented skin, Nigerians who spend several hours day outdoors are making vitamin D that impacts their blood serum levels. While this article does not provide positive proof that Onifade's test patients spent 8 plus hours a day outdoors and also had adequate serum levels of vitamin D, this article does show that other Nigerians tested were converting sunlight into vitamin D at adequate levels.

[The abstract below states that a calcium deficiency, not a vitamin D deficiency, is what contributes to rickets in Nigerian children]

Absence of vitamin D deficiency in young Nigerian children.

J Pediatr. 1998 Dec;133(6):740-4. Pfitzner MA1, Thacher TD, Pettifor JM, Zoakah AI, Lawson JO, Isichei CO, Fischer PR.

Abstract OBJECTIVE: To determine the prevalence of vitamin D deficiency in young Nigerian children residing in an area where nutritional rickets is common.

STUDY DESIGN: A randomized cluster sample of children aged 6 to 35 months in Jos, Nigeria.

RESULTS: Of 218 children evaluated, no child in the study had a 25-hydroxyvitamin D (25-OHD) concentration <10 ng/mL (the generally held definition of vitamin D deficiency). Children spent an average of 8.3 hours per day outside of the home. Twenty children (9.2%) had clinical findings of rickets. Children with clinical signs of rickets were more likely to be not currently breast fed and have significantly lower serum calcium concentrations than those without signs of rickets (9.1 vs 9.4 mg/dL, respectively, P =.01). Yet, 25-OHD levels were not significantly different between those children with clinical signs of rickets and those without such clinical signs.

CONCLUSION: Vitamin D deficiency was not found in this population of young children in whom clinical rickets is common. This is consistent with the hypothesis that dietary calcium insufficiency, without preexisting vitamin D deficiency, accounts for the development of clinical rickets in Nigerian children.

Note: I am including this article because of an email I received this morning in response to yesterday's postings on this website on my theory that sunlight and adequate vitamin D levels in Onifade's test patients helped their immune system keep HIV viral loads under 50K and that vitamin D levels resulting from exposure to the sun was a co-factor with the use of Black Seed in the experiments impressive results.

Study finds Vitamin D levels low in HIV + patients

A study done at the University of California Los Angeles (UCLA) by JE Lake MD and JS Adams, MD was published in Curr HIV/AIDS Rep. 2011 September; 8(3): 133-141. Doi:10.1007/s11904-011-0082-8

The researchers (Adams and Lake) found that vitamin D levels are low in both the general population and in the HIV subset group. They also found some HIV drugs (AZT, efavirenz and tenofovir) were linked to lower vitamin D levels in patients with HIV. Because of this, HIV patients were at an even greater risk for vitamin D deficiency when using these drugs. One drug, ritonavir, was found to have a protective effect against vitamin D depletion.

The authors also said that studies are needed on all other HIV drugs to determine their effects on vitamin D serum levels. To date, these studies have not been done. Vitamin D levels and CD4 counts The authors stated that in reference to vitamin D and CD4 counts: “most studies have shown a positive association” They referred to the following five studies -

1. Villamor E. A potential role for vitamin D on HIV infection? Nutr Rev. May; 2006 64(5 Pt 1):226– 33. [PubMed: 16770943]

. Haug C, Muller F, Aukrust P, Froland SS. Subnormal serum concentration of 1,25-vitamin D in human immunodeficiency virus infection: correlation with degree of immune deficiency and survival. J Infect Dis. Apr; 1994 169(4):889–93. [PubMed: 7907645]

3. Haug CJ, Aukrust P, Haug E, Morkrid L, Muller F, Froland SS. Severe deficiency of 1,25- dihydroxyvitamin D3 in human immunodeficiency virus infection: association with immunological hyperactivity and only minor changes in calcium homeostasis. J Clin Endocrinol Metab. Nov; 1998 83(11):3832–8. [PubMed: 9814454]

4. Teichmann J, Stephan E, Lange U, Discher T, Friese G, Lohmeyer J, et al. Osteopenia in HIV- infected women prior to highly active antiretroviral therapy. J Infect. May; 2003 46(4):221–7. [PubMed: 12799147]

5. de Luis DA, Bachiller P, Aller R, de Luis J, Izaola O, Terroba MC, et al. Relation among micronutrient intakes with CD4 count in HIV infected patients. Nutr Hosp. Nov-Dec;2002 17(6): 285–9. [PubMed: 12514921] Study in Spain finds an increase in CD4 cells for each mcg of vitamin D intake Nutr Hosp. 2002 Nov-Dec;17(6):285-9.

Relation among micronutrient intakes with CD4 count in HIV infected patients. [Article in Spanish]

Authors - de Luis DA1, Bachiller P, Aller R, de Luis J, Izaola O, Terroba MC, Cuellar L, González Sagrado M.

"INTRODUCTION: The impact of micronutrients on HIV disease progression has been an area of great interest. Several studies have shown an association between disease progression and micronutrient status. The aim of our study was to assess the correlation between micronutrients intakes and immune status in HIV infected patients.

"MATERIAL AND METHODS: A total of 119 patients were evaluated. Nobody dropped out. In all patients the following parameters were assessed; age, sex, treatment with anti-retroviral drugs, performed an anthropometric evaluation (weight, tricipital skinfold, midarm circumference, and body mass index (BMI)) and a biochemical evaluation (glucose, albumin, prealbumin, transferrin, total proteins, lymphocytes and count of CD4). All patients received instruction in 24-hour written food record keeping.

"RESULTS: Patients had an average age 37.9 +/- 9.9 years, weight 64.5 +/- 13.2 kg and body mass index 22.5 +/- 3.5. Levels of total proteins, albumin, prealbumin y transferrin were normal. Percentile distribution of anthropometric parameters showed a deep depletion in muscular protein compartment, 53.1% of patients had tricipital skinfold under P 50, 91.8% had midarm muscle circumference under P 50. The correlation analysis among dietary intake and immune status, showed a positive association among vitamin A intake and vitamin D with CD4+ (r = 0.35; p < 0.01) and (r = 0.51; p < 0.001), respectively.

In the multivariant analysis with dependent variable (CD4 count), only vitamin D remained in the model (F = 16.99; p < 0.001), with an increase of 34 (CI 95%: 5.81-167.3) CD4+ (count/uL) with each microgram of vitamin D intake, adjusted by age, sex, energy and protein intake, and anti-retroviral drugs.

CONCLUSION: Vitamin A, and D intakes were correlated with CD4 count, only vitamin D remained as an independent predictor parameter in a multivariant model."

How much is a microgram (mcg) of vitamin D?

The previous article stated that for each microgram of vitamin D used, the CD4 count increased. That is very impressive, but how do you translate i.u. (international units) into micrograms? In other words, how many mcg are there in 4000 I.U. of vitamin D? The answer, if I calculated this correctly, is 100 mcg or 1/10th of one milligram.

Suggestions on how to increase Vitamin D levels

Now, here are a few things to consider doing now and taking notes on:

1. If you are under a doctor's case, ask for a blood serum test of vitamin D levels and while you are at it, ask for selenium levels as well. If you have AIDs or cancer, you want these numbers to be on the upper end of the reference range to maximize their immune enhancing and anti-inflammatory benefits.

2. If you cannot get one hour or more of daily exposure to the sun from 11 am to 2 pm, then consider 15 to 20 minutes using a sun tanning bed 3 to 5 times a week, and/or, take Vitamin D3 as a supplement at 5000 to 10,000 i.u daily while your doctor monitors your blood levels.

Take adequate magnesium (500 to 1000 mg daily) to insure proper utilization of the Vitamin D. Other co-factors widely reported that help utilize vitamin D and prevent side effects from very high doses are vitamin A and vitamin K. Vitamin K is found in parsley and other green leafy vegetables. Moderate doses of vitamn D3 in adults are in the 5,000 to 10,000 i.u daily. High doses are 25,000 to 100,000 i.u daily. For most purposes, 30 to 40 ug/ml in blood tests are the initial target range to increase vitamin D3 levels. According to Jeff Bowles in his book (see last page of this newsletter) about his personal experiments with high doses of vitamin D3, adverse effects do not occur until blood levels exceed 100 ug/ml.

3. Keep track to determine if a rise in blood levels of vitamin D3 (and selenium) correlate to an increase in CD4 counts and a decrease in the viral load while using Black Seed or prescribed meds as a remedy.

Note - you can increase selenium levels by eating 4 to 8 Brazil nuts daily - they are the world's richest source of selenium.

My personal experience with psoriasis, outdoors in the sun, indoor tanning, and vitamin D supplements

Conrad LeBeau

The heartbreak of psoriasis has affected me all my life from my teenage years living on a farm in the Upper Pennsylvania of Michigan in the 1950’s to life as a city urban dweller since 1960. For me, psoriasis has been limited to scaly white patches on my elbows and knees and not the red itchy inflamed patches sometimes seen.

As I recall in the good ole summertime on the small dairy farm where I grew up west of Carney, Michigan, the psoriasis would disappear in the summer and return in the winter. I recall also taking cod liver oil at the time and it seemed to help.

I also found that taking lecithin daily and restricting fat intake reduced the psoriasis to almost non-existence. The problem is that there has never been a cure. My lifelong addiction to dairy ice cream has been a double whammy – promoting both the growth of psoriasis and recurring colds and frequent sinus infections.

I have consistently found that spending more time outdoors in the sun improved my sense of well being and also did wonders to help me get a good night’s sleep. For the past 25 years or so I have, from time to time, used a tanning bed 2 or 3 times a week. I start with a daily exposure of 15 minutes and work up to 20 minutes. I have never used sun tanning lotion or oils as I am not convinced they are safe and they block most of the UVB light that causes vitamin D to be produced in the skin. UVB may have other beneficial effects as well.

Of course I always use the protective UV blocking eyewear to prevent eye damage from direct over-exposure to UV light. For the same reason, you don’t directly look at the sun when you are outdoors.

In March, 2015, I stopped using the tanning bed at the local athletic club that I belonged to, and decided to try direct tanning from the sun. Unfortunately, I had to do this indoors using the sunlight passing through a window at the gym. [I only recently learned that sunlight passing through glass filters out the UVB rays. These are the very rays you need to reach your skin to produce vitamin D]

Due to cloudy skies, and the filtration of the light rays by the window, my skin was not tanning. At the time I also was not taking any vitamin D supplements. By the end of May, I had developed a fairly serious sinus infection and even with my best home remedies, I concluded I needed to see an allergist or immunologist.

I have original Medicare and found an allergist in the local Yellow Page phone directory who advertised treating sinus infections. When I called I was told they took Medicare so I did not need to first see a primary physician first to get a referral as is often required when you use an HMO or Advantage plan.

On June 4th, I arrived for the evaluation. Ironically, because of those home remedies and the complete elimination of all milk products my bronchial congestion, sinuses and breathing had substantially improved. The doctor spent at least one hour with me, and his evaluation was very thorough. He did the skin prick test for environmental allergens. He tested for 40 different possible allergens from cats to dogs to grass, pollens and leaves, etc. He found that I had a very high reaction to dust mites, as high as my reaction was to ragweed. Both dust mites on a scale of 1 to 5 were at a 5. Everything else was less - usually from a 1 to a 3.

The reaction was determined by the size of the welt that formed within 5 to 10 minutes after the skin prick test. The skin prick test with the foreign antigens were done with 20 on each arm. He took the time to explain how to dust mite proof my bedroom, and because he also determined the original cold virus that caused my sinus infection may have become a bacterial co-infection, he prescribed a 10-day regimen of antibiotics.

Combining the steps taken to dust mite proof my bedroom, the antibiotics, and the diet modifications, the use of a topical hydrogen peroxide gel, and various herbal remedies, I was cleared of the infection in about one week. I was now able to sleep again, not as a mouth breather, but could breathe normally through my nose. Because millions of people are sensitive to dust mites, I have described below what I done.

How I dust mite proofed my bedroom

Dust mites live in dust, eat dead skin that flakes off our bodies, and are found in mattresses, pillows, blankets, and upholstered furniture. Once in awhile it pays to throw out all your pillows and get new ones. However, you also need dust mite proof pillow covers and a dust mite proof mattress cover. I found what I needed at Kmart. I bought a dust mite proof mattress cover for about $30 and dust mite pillow covers for less than $10 each. I also bought a pillow that is pre-shaped like an hourglass to support the neck and head.

I was also advised to obtain a room air purifier with a Hepa filter. I did all that, and I can say it was well worth it. Avoidance is one way to reduce the adverse effects of an offending substance. Immuno-therapy is another way to desensitize the immune system and reduce inflammatory reactions. Finally when I left the office I asked how much I owed and they said nothing.

This small clinic does not charge co-pays and is satisfied with what they collect from Medicare. If I had been in an HMO plan or a Medicare Advantage Plan, they would have been asking for a co-pay from the get go. For me the advantage is to stay independent and use original Medicare.

63,726 Scientific Articles on Vitamin D

63,726 scientific articles on the one subject of vitamin D and your health could keep the most avid speed-reader busy for several months if not several years. A scan of the published literature finds that vitamin D deficiency is linked to all the following health conditions and diseases. This is a partial list -

1. Insomnia

2. Cancer – all types

3. High blood pressure

4. Cardiovascular disease

5. Multiple Sclerosis

6. Depression

7. Low melatonin

8. Obesity

9. Kidney disease

10. Diabetes type 2

11. Retinopathy – damage to retina

12. Chronic infections

13. Hepatitis B

14. Rheumatism

15. Autoimmune disease

16. Osteoporosis – bone density

17. Candidiasis and fungal overgrowth

Sun Screen blocks most UVB radiation and reduces production of vitamin D

The publicity drums beat loudly to protect you from dangerous UV rays from the sun. Fear the sun, they say, and add that the suns UVB rays may cause you to develop melanoma, a deadly form of skin cancer. However, what they don’t tell you is that the UVB rays turn on the skins production of vitamin D, a hormone that protects you from cancer.

Scientific studies have found that persons with melanoma are deficient or who have low levels of vitamin D (1, 2). Since vitamin D has been shown to protect against many forms of cancer (3,4,5), could melanoma actually be caused by a lack of sunlight reaching the skin? In other words, would more sun tanning outdoors and indoors help to prevent future skin cancer and melanomas by increasing blood serum levels of vitamin D? Melanomas may further develop from the regular use of sunscreen or sun tanning lotion that blocks UVB rays from reaching the skin.

1. PLoS One. 2015 May 13;10(5):e0126394. doi: 10.1371/journal.pone.0126394. eCollection 2015. Vitamin D deficiency at melanoma diagnosis is associated with higher breslow thickness. Authors: Wyatt C1, Lucas RM2, Hurst C3, Kimlin MG1.

2. Int J Cancer. 2015 Jun 15;136(12):2890-9. doi: 10.1002/ijc.29334. Epub 2014 Dec 13. 25-Hydroxyvitamin D2 /D3 levels and factors associated with systemic inflammation and melanoma survival in the Leeds Melanoma Cohort. Newton-Bishop JA1, Davies JR, Latheef F, Randerson-Moor J, Chan M, Gascoyne J, Waseem S, Haynes S, O'Donovan C, Bishop DT.

3. Vitamin D3 enhances antitumor activity of metformin in human bladder carcinoma SW-780 cells. Guo LS, Li HX, Li CY, Zhang SY, Chen J, Wang QL, Gao JM, Liang JQ, Gao MT, Wu YJ. Pharmazie. 2015 Feb;70(2):123-8.

4. 25-Dihydroxyvitamin D3 Inhibits Esophageal Squamous Cell Carcinoma Progression by Reducing IL6 Signaling. Chen PT, Hsieh CC, Wu CT, Yen TC, Lin PY, Chen WC, Chen MF. Mol Cancer Ther. 2015 Jun;14(6):1365-75. doi: 10.1158/1535-7163.MCT-14-0952. Epub 2015 Mar 30.

5. Activation of vitamin D receptor signaling down regulates the expression of nuclear FOXM1 protein and suppresses pancreatic cancer cell stemness. Li Z, Jia Z, Gao Y, Xie D, Wei D, Cui J, Mishra L, Huang S, Zhang Y, Xie K. Clin Cancer Res. 2015 Feb 15;21(4):844-53. doi: 10.1158/1078-0432.CCR-14-2437.

6. 25-dihydroxyvitamin D3 inhibits cell growth and NFKB signaling in tamoxifen-resistant breast cancer cells. Lundqvist J, Yde CW, Lykkesfeldt AE. Steroids. 2014 Jul;85:30-5. doi: 10.1016/j.steroids.2014.04.001.

Classical and emerging roles of vitamin D in hepatitis C virus infection.

Semin Liver Dis. 2011 Nov;31(4):387-98. doi: 10.1055/s-0031-1297927. Gutierrez JA1, Parikh N, Branch AD.

“In keeping with the results of randomized clinical trials, many observational studies have demonstrated an association between higher 25(OH)D levels and reduced all-cause mortality and reduced disease-specific mortality, including mortality due to renal disease, cardiovascular disease, and cancer 49-51. A recent analysis of NHANES III data showed that adults over the age of 65 years with serum 25(OH)D levels greater than 40 ng/mL had a 45% lower risk of death than those with 25(OH)D less than 10 ng/mL”

In the above full text article, the authors quoted the target range of vitamin D serum levels inpatients with HCV should be 32 ng/ml or higher. In searching for optimal vitamin D levels in the general population they wrote:

“Available data suggest that the optimal level of 25(OH) for good health in the general population is about 35 ng/ml. As discussed in the opening section of this article, some experts advise higher levels, but none considers 35 ng/mL to exceed the optimal range. In the absence of outcomes data in HCV-positive patients, 35 ng/mL of 25(OH)D is the best target level to aim for. This can be achieved in almost all liver disease patients through the use of oral supplements.”

Books on Vitamin D

I recently located and purchased 3 books on Vitamin D. Two of the books are written by medical doctors, Michael Holick, and the other by Sarfraz Zaidi. The 3rd book is by Jeff Bowles, a self-educated individual. I have not yet read or reviewed the two books by the medical doctors who both favor the use of vitamin D in the prevention and treatment of many health problems. I will study their books and report on what I find in the next Journal of Immunity.

I read Jeff Bowles book titled “The Miraculous Results of Extremely High Doses of Vitamin D3.” Bowles used doses up to 100,000 i.u daily of vitamin D3 over a one year period. He stated:

“Vitamin D 3 therapy over the last year cured all my chronic conditions – some that I’d had for 20+ years” His personal claims include -

1. A painful snapping hip syndrome which I had been suffering from for 23 years and no Dr. could help me – It is now 100% gone.

2. Yellow fungus infected toenails. I tried everything for 20 years and nothing worked – 10 months of high dose vitamin D and they are clear as a bell!

3. A knobby bone spur on my elbow that made me look like Popeye the sailor man. It is now 100% dissolved and my elbow is back to the way it used to be 20 years ago.

4. Painful, stiff Arthritic shoulders that prevented me fro even throwing a ball from home plate…I can now throw the ball twice as far.

5. A ganglion cyst that persisted on my wrist for over 5 years has shrunk from the size of a golf ball to the size of a pea and now it is rock hard, painless, and shrinking.

6. A small subcutaneous cyst on my face that had not gone away for 20 years – now gone!

7. Without even trying my weight has dropped by 25 lbs for 204 to 179. Bowles also discusses the use of magnesium and vitamin K and added that these should be taken when very high vitamin D doses are used. The book is self published and marketed on the internet and at"

Vitamin D - How much is too much?

Clin Endocrinol (Oxf). 2015 Jun 6. doi: 10.1111/cen.12836. Vitamin D toxicity resulting from overzealous correction of vitamin D deficiency. by Kaur P1, Mishra SK1, Mithal A1.

The Abstract reported on women in India who were consuming more than 1,000,000 i.u of vitamin D daily and doing this continuously over a period of time. One million (1,000,000) i.u. of vitamn D would be the equivalent of swallowing 100 capsules each of which contain 10,000 i.u. of vitamin D. Taking 100 vitamin D capsules each day would be an extremely high dose and is not recommended. Here is what the abstract said:

“Vitamin D toxicity, often considered rare, can be life-threatening and associated with substantial morbidity, if not identified promptly.” The patients who had symptoms of vitamin D toxicity, were evaluated between January 2011 and January 2013 in Delhi-NCR, India.

“Evaluation included detailed clinical history and biochemical tests including serum calcium, phosphorus, creatinine, intact parathyroid hormone and 25 hydroxyvitamin D (25(OH)D).”

Findings: “Sixteen patients with vitamin D toxicity were seen. Clinical manifestations included nausea, vomiting, altered sensorium, constipation, pancreatitis, acute kidney injury and weight loss.”

Note: Jeff Bowles in his book stated that his research finds that adverse effects from high doses of vitamin D do not start until blood serum levels exceed 100 ug/ml. He also stated that life guards in southern Florida typically have vitamin D levels around 120 ug/ml from sun exposure alone, and without any side effects. This is in contrast to millions of Americans with cancer, AIDS, auto immune diseases, obesity, high blood pressure, type II diabetes, multiple sclerosis, chronic infections, and other conditions mentioned earlier whose vitamin D levels are typically less than 10 - a level that does not begin to meet the average persons minimum needs for vitamin D. Several sources are also claiming that vitamin D is a misnomer and is actually a hormone.

FDA vs. LeBeau update

After waiting 2 plus years for a decision upon his review, Federal Judge Charles Clevert has set a date of July 21 at 3 pm for oral arguments in the case of 10-CR-00253. The hearing will be in Room 222 at the Federal Courthouse 517 E Wisconsin Ave, Milwaukee, Wisconsin 53202.

I will be making the oral arguments in this case with standby counsel. This is an appeal from a decision of Magistrate Callahan from 2012 in which I am seeking to bring the FDA into compliance with the U.S. Constitution, the original definition of “drug” from the FDC Act of 1906, and the intent of Congress in passing the Dietary Health Supplement and Education Act of 1994 (DHSEA). The public may attend and if you live in southeastern Wisconsin, I would encourage you to do so to show your support for health freedom rights. Thank you for your prayers and support. Conrad LeBeau

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